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Triacsin C

Cat. No. 2472 1 次引用 提交评论 说明书 / 分析证书（CoA） / 化学品安全技术说明书（SDS）
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说明： Inhibitor of acyl-CoA synthetase
化学名： (2E,4E,7E)-2,4,7-Undecatrienal nitrosohydrazone
纯度： ≥95% (HPLC)
说明书
引用文献 (1)
评论
生物活性 技术数据 溶解性 计算器 说明书 参考文献

生物活性 for Triacsin C

Triacsin C is an inhibitor of acyl-CoA synthetase. Inhibits lipid synthesis and cell proliferation in Raji lymphoma cells. Induces apoptosis in cancer cell lines and inhibits human lung cancer cell xenograft tumor growth in mice.

技术数据 for Triacsin C

分子量 207.27
公式 C11H17N3O
储存 Store at -20°C
纯度 ≥95% (HPLC)
CAS Number 76896-80-5
PubChem ID 9576787
InChI Key NKTGCVUIESDXPU-YLEPRARLSA-N
Smiles O=NN/N=C/C=C/C=C/C/C=C/CCC

上方提供的技术数据仅供参考。批次相关数据请参见分析证书。

Tocris products are intended for laboratory research use only, unless stated otherwise.

溶解性数据 for Triacsin C

溶剂 最高浓度 mg/mL 最高浓度 mM
溶解性
DMSO 5 25

制备储备液 for Triacsin C

以下数据基于产品分子量 207.27。 Batch specific molecular weights may vary from batch to batch due to the degree of hydration, which will affect the solvent volumes required to prepare stock solutions.

选择批次从而根据批次分子量重新计算:
浓度/溶剂体积/质量 1 mg 5 mg 10 mg
0.25 mM 19.3 mL 96.49 mL 192.98 mL
1.25 mM 3.86 mL 19.3 mL 38.6 mL
2.5 mM 1.93 mL 9.65 mL 19.3 mL
12.5 mM 0.39 mL 1.93 mL 3.86 mL

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产品说明书 for Triacsin C

分析证书/产品说明书
选择另一批次：
化学品安全技术说明书

参考文献 for Triacsin C

参考文献是支持产品生物活性的出版物。

Tomoda et al (1991) Evidence for an essential role of long chain acyl-CoA synthetase in animal cell proliferation. Inhibition of long chain acyl-CoA synthetase by triacsins caused inhibition of Raji cell proliferation. J.Biol.Chem. 266 4214 PMID: 1999415

Mashima et al (2005) p53-defective tumors with a functional apoptosome-mediated pathway: a new therapeutic target. J.Natl.Cancer.Inst. 97 765 PMID: 15900046

If you know of a relevant reference for Triacsin C, please let us know.

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关键词: Triacsin C, Triacsin C supplier, Triacsin, C, inhibitors, inhibits, acyl-coa-synthetase, antitumor, Other, Synthases/Synthetases, 2472, Tocris Bioscience

1 篇 Triacsin C 的引用文献

引用文献是使用了 Tocris 产品的出版物。 Triacsin C 的部分引用包括：

Lancaster et al (2018) Evidence that TLR4 Is Not a Receptor for Saturated Fatty Acids but Mediates Lipid-Induced Inflammation by Reprogramming Macrophage Metabolism. Cell Metab 27 1096 PMID: 29681442

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