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Submit Review3-Aminobenzamide
说明： PARP inhibitor
别名： 3-ABA
生物活性 for 3-Aminobenzamide
3-Aminobenzamide is an inhibitor of poly(ADP-ribose) polymerase, which protects cells from UV-B-induced apoptosis via influence on the cytoskeleton.
技术数据 for 3-Aminobenzamide
| 分子量 | 136.15 |
| 公式 | C7H8N2O |
| 储存 | Store at RT |
| CAS Number | 3544-24-9 |
| PubChem ID | 1645 |
| InChI Key | GSCPDZHWVNUUFI-UHFFFAOYSA-N |
| Smiles | NC(=O)C1=CC=CC(N)=C1 |
上方提供的技术数据仅供参考。批次相关数据请参见分析证书。
Tocris products are intended for laboratory research use only, unless stated otherwise.
产品说明书 for 3-Aminobenzamide
参考文献 for 3-Aminobenzamide
参考文献是支持产品生物活性的出版物。
Cuzzocrea et al (1997) Beneficial effects of 3-aminobenzamide, an inhibitor of poly (ADP-ribose) synthetase in a rat model of splanchnic artery occlusion and reperfusion. Br.J.Pharmacol. 121 1065 PMID: 9249240
Malorni et al (1995) 3-Aminobenzamide protects cells from UV-B-induced apoptosis by acting on cytoskeleton and substrate adhesion. Biochem.Biophys.Res.Commun. 207 715 PMID: 7864864
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关键词: 3-Aminobenzamide, 3-Aminobenzamide supplier, PARP, inhibitors, inhibits, PolyADP-ribose, Polymerases, 3-ABA, Poly(ADP-ribose), Polymerase, 0788, Tocris Bioscience
1 篇 3-Aminobenzamide 的引用文献
引用文献是使用了 Tocris 产品的出版物。 3-Aminobenzamide 的部分引用包括：
Pakula et al (2019) The Epithelial-Mesenchymal Transition Initiated by Malignant Ascites Underlies the Transmesothelial Invasion of Ovarian Cancer Cells. Int J Mol Sci 20 PMID: 30609691
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该领域的文献
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Cell Cycle and DNA Damage Research Product Guide
This product guide provides a review of the cell cycle and DNA damage research area and lists over 150 products, including research tools for:
- Cell Cycle and Mitosis
- DNA Damage Repair
- Targeted Protein Degradation
- Ubiquitin Proteasome Pathway
- Chemotherapy Targets
Cell Cycle & DNA Damage Repair Poster
In normal cells, each stage of the cell cycle is tightly regulated, however in cancer cells many genes and proteins that are involved in the regulation of the cell cycle are mutated or over expressed. This poster summarizes the stages of the cell cycle and DNA repair. It also highlights strategies for enhancing replicative stress in cancer cells to force mitotic catastrophe and cell death.