SAMS Peptide

Discontinued Product

1344 has been discontinued.

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说明: AMP-activated protein kinase substrate
说明书
引用文献 (1)
评论
通路 (1)

生物活性 for SAMS Peptide

SAMS Peptide is a substrate for AMP-activated protein kinase (AMPK). Phosphorylated rapidly by AMPK, it is more specific for the kinase than acetyl CoA carboxylase itself, and provides a convenient and sensitive assay for AMPK.

技术数据 for SAMS Peptide

分子量 1779.15
公式 C74H131N29O18S2
序列 HMRSAMSGLHLVKRR
储存 Desiccate at -20°C
CAS Number 125911-68-4
PubChem ID 71773161
InChI Key YJAHUEAICBOBTC-CSVPVIMBSA-N
Smiles [H]N[C@@H](CC1=CNC=N1)C(=O)N[C@@H](CCSC)C(=O)N[C@@H](CCCNC(N)=N)C(=O)N[C@@H](CO)C(=O)N[C@@H](C)C(=O)N[C@@H](CCSC)C(=O)N[C@@H](CO)C(=O)NCC(=O)N[C@@H](CC(C)C)C(=O)N[C@@H](CC1=CNC=N1)C(=O)N[C@@H](CC(C)C)C(=O)N[C@@H](C(C)C)C(=O)N[C@@H](CCCCN)C(=O)N[C@@H](CCCNC(N)=N)C(=O)N[C@@H](CCCNC(N)=N)C(O)=O

上方提供的技术数据仅供参考。批次相关数据请参见分析证书。

Tocris products are intended for laboratory research use only, unless stated otherwise.

产品说明书 for SAMS Peptide

分析证书/产品说明书
选择另一批次:

参考文献 for SAMS Peptide

参考文献是支持产品生物活性的出版物。

Carling et al (1989) Purification and characterization of the AMP-activated protein kinase. Eur.J.Biochem. 186 129 PMID: 2598924

Davies et al (1989) Tissue distribution of the AMP-activated protein kinase, and lack of activation by cyclic-AMP-dependent protein kinase, studied using a specific and sensitive peptide assay. Eur.J.Biochem. 186 123 PMID: 2574667

Hardie et al (1998) The AMP-activated/SNF1 protein kinase subfamily: metabolic sensors of the eukaryotic cell? Annu.Rev.Biochem 67 821 PMID: 9759505

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关键词: SAMS Peptide, SAMS Peptide supplier, AMP-activated, protein, kinase, AMPK, Kinases, Enzyme, Substrates, Activators, /, 1344, Tocris Bioscience

1 篇 SAMS Peptide 的引用文献

引用文献是使用了 Tocris 产品的出版物。 SAMS Peptide 的部分引用包括:

Thelander et al (2004) Snf1-related protein kinase 1 is needed for growth in a normal day-night light cycle. EMBO J 23 1900 PMID: 15057278


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Pathways for SAMS Peptide

AMPK Signaling Pathway

AMPK Signaling Pathway

AMPK signaling pathway plays an important role in the cellular reponse to low levels of available ATP, often caused by stresses such as heat shock, ischemia or hypoxia.